Since no one has responded to your question, I'll put my two cents in and say no, calcifications are not evidence of heart disease. I think Dr. Mandrola's dislike for the test is precisely because we don't know what the results mean. Why do some people with atherosclerosis develop calcifications and others don't? Who knows? Might the cal…
Since no one has responded to your question, I'll put my two cents in and say no, calcifications are not evidence of heart disease. I think Dr. Mandrola's dislike for the test is precisely because we don't know what the results mean. Why do some people with atherosclerosis develop calcifications and others don't? Who knows? Might the calcifications protect the arterial intima from rupture rather than make it more likely? Nobody knows. What is certain is that multiple studies have shown that aspirin has no significant effect in preventing myocardial infarction or other cardiovascular events in either primary or secondary therapy.
I appreciate you responding. My understanding is different. While the relationship between CAC and plaque susceptibility is not completely understood, the presence of CAC is direct evidence of the presence of coronary atherosclerosis. This seems universally agreed upon in the litterature. What to do about it is another question.
Since no one has responded to your question, I'll put my two cents in and say no, calcifications are not evidence of heart disease. I think Dr. Mandrola's dislike for the test is precisely because we don't know what the results mean. Why do some people with atherosclerosis develop calcifications and others don't? Who knows? Might the calcifications protect the arterial intima from rupture rather than make it more likely? Nobody knows. What is certain is that multiple studies have shown that aspirin has no significant effect in preventing myocardial infarction or other cardiovascular events in either primary or secondary therapy.
I appreciate you responding. My understanding is different. While the relationship between CAC and plaque susceptibility is not completely understood, the presence of CAC is direct evidence of the presence of coronary atherosclerosis. This seems universally agreed upon in the litterature. What to do about it is another question.